Abstract
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) interacts between the host and virus and govern induction, resulting in multiorgan impacts. Its pathophysiology involves the followings: 1) the angiotensin-converting enzyme (ACE2) and Toll-like receptor (TLR) pathways: 2) the neuropilin (NRP) pathway: 3) the spike protein pathway. Therefore, it is necessary to block the pathological course with modulating innate lymphoid cells against diverse corona variants in the future.
Original language | English |
---|---|
Article number | 100766 |
Number of pages | 11 |
Journal | Biomedical Journal |
Early online date | 14 Jul 2024 |
DOIs | |
Publication status | E-pub ahead of print - 14 Jul 2024 |
Bibliographical note
Copyright © 2024 The Authors. Published by Elsevier B.V. on behalf of Chang Gung University. This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License (https://creativecommons.org/licenses/by-nc-nd/4.0/).Keywords
- COVID-19
- TLR
- SARS-CoV-2
- ACE2
- Spike protein