Mechanism of attenuation of protein loss in murine C₂C₁₂ myotubes by d-myo-inositol 1,2,6-triphosphate

Keyphrases

• Angiotensin II (Ang II) 66%
• Attenuation 100%
• Autophosphorylation 33%
• C2C12 Myotubes 100%
• Cachexia Models 33%
• Caspase-3 33%
• Catabolic 66%
• Chelates 33%
• Divalent Metal Ions 33%
• Double-stranded RNA 33%
• Downstream Signaling 33%
• EIF2 33%
• Increased Activity 33%
• Inositol 100%
• Interferon-α (IFN-α) 33%
• Lipopolysaccharide 33%
• Muscle Atrophy 66%
• Myo-inositol 100%
• Myotubes 33%
• Phosphorylation 33%
• Protein Degradation 100%
• Protein Kinase PKR 33%
• Protein Loss 100%
• Protein Synthesis 100%
• Proteolysis-inducing Factor 100%
• Signaling Pathway 33%
• Triphosphate 100%
• Tumor Necrosis Factor-α 33%
• Ubiquitin-proteasome System 66%
• Zn2+ 33%

Biochemistry, Genetics and Molecular Biology

• Angiotensin 66%
• Autophosphorylation 33%
• C2C12 100%
• Caspase 3 33%
• Double-Stranded RNA 33%
• Eukaryotic Initiation Factor 33%
• Inositol 100%
• Interferon 33%
• Lipopolysaccharide 33%
• Murine 100%
• Proteasome 66%
• Protein Biosynthesis 100%
• Protein Catabolism 100%
• Protein Kinase R 33%
• Proteolysis 100%
• Tumor Necrosis Factor 33%
• Ubiquitin 66%

Neuroscience

• Angiotensin II 25%
• Cachexia 12%
• Caspase 3 12%
• Inositol 100%
• Inositol 1,2,6 Trisphosphate 100%
• Interferon 12%
• Lipopolysaccharide 12%
• Muscle Atrophy 25%
• Necrosis 12%
• Proteasome 25%
• Protein Biosynthesis 37%
• Protein Kinase R 12%
• Proteolysis 37%
• Prothrombin 25%
• Ubiquitin 25%