Nitric oxide-dependent bone marrow progenitor mobilization by carbon monoxide enhances endothelial repair after vascular injury

Barbara Wegiel, David J. Gallo, Kathleen G. Raman, Jenny M. Karlsson, Brett Ozanich, Beek Y. Chin, Edith Tzeng, Shakil Ahmad, Asif Ahmed, Catherine J. Baty, Leo E. Otterbein

Research output: Contribution to journalArticle

Abstract

Carbon monoxide (CO) has emerged as a vascular homeostatic molecule that prevents balloon angioplasty-induced stenosis via antiproliferative effects on vascular smooth muscle cells. The effects of CO on reendothelialization have not been evaluated.
Original languageEnglish
Pages (from-to)537-548
Number of pages12
JournalCirculation
Volume121
Issue number4
DOIs
Publication statusPublished - Feb 2010

Bibliographical note

© 2010 American Heart Association, Inc.

Keywords

  • angioplasty
  • endothelium
  • nitric oxide synthase
  • signal transduction
  • stenosis

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    Wegiel, B., Gallo, D. J., Raman, K. G., Karlsson, J. M., Ozanich, B., Chin, B. Y., Tzeng, E., Ahmad, S., Ahmed, A., Baty, C. J., & Otterbein, L. E. (2010). Nitric oxide-dependent bone marrow progenitor mobilization by carbon monoxide enhances endothelial repair after vascular injury. Circulation, 121(4), 537-548. https://doi.org/10.1161/CIRCULATIONAHA.109.887695