Carbon monoxide (CO) has emerged as a vascular homeostatic molecule that prevents balloon angioplasty-induced stenosis via antiproliferative effects on vascular smooth muscle cells. The effects of CO on reendothelialization have not been evaluated.
Bibliographical note© 2010 American Heart Association, Inc.
- nitric oxide synthase
- signal transduction
Wegiel, B., Gallo, D. J., Raman, K. G., Karlsson, J. M., Ozanich, B., Chin, B. Y., Tzeng, E., Ahmad, S., Ahmed, A., Baty, C. J., & Otterbein, L. E. (2010). Nitric oxide-dependent bone marrow progenitor mobilization by carbon monoxide enhances endothelial repair after vascular injury. Circulation, 121(4), 537-548. https://doi.org/10.1161/CIRCULATIONAHA.109.887695