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Obesity in the pathogenesis of type 2 diabetes
Caroline Day,
Clifford J. Bailey
School of Biosciences
Aston Research Centre for Health in Ageing
College of Health and Life Sciences
Research output
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Article
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peer-review
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Dive into the research topics of 'Obesity in the pathogenesis of type 2 diabetes'. Together they form a unique fingerprint.
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Keyphrases
Obesity
100%
Type 2 Diabetes Mellitus (T2DM)
100%
Insulin Resistance
66%
Liver
33%
Detrimental Effects
33%
Protein Kinase
33%
Hyperglycemia
33%
Adipose Tissue
33%
Ceramide
33%
Fatty Acids
33%
Islet β-cell Function
33%
Insulin Signaling
33%
Adiponectin
33%
Cellular Energy
33%
Adipokines
33%
Diacylglycerol
33%
Lipid Levels
33%
Visceral Adipose Tissue
33%
Adipose Mass
33%
Circulating Fatty Acids
33%
Retinol-binding Protein 4 (RBP4)
33%
Biochemistry, Genetics and Molecular Biology
Insulin Resistance
100%
Metabolite
50%
calcium activated,phospholipid dependent protein kinase
50%
Hyperglycemia
50%
Ceramide
50%
Cell Function
50%
Isoform
50%
Adiponectin
50%
Tumor Necrosis Factor
50%
Adipokine
50%
Diglyceride
50%
Lipid Level
50%
Insulin Signaling
50%
Cell Energy
50%
Medicine and Dentistry
Maturity Onset Diabetes of the Young
100%
Adipose Tissue
66%
Insulin Resistance
66%
Fatty Acid
66%
Tumor Necrosis Factor
33%
Cell Function
33%
Obesity
33%
Hyperglycemia
33%
Insulin Signaling
33%
Beta Cell
33%
Adipocytokine
33%
Diacylglycerol
33%
Cell Energy
33%
Ceramide
33%
Adiponectin
33%
Metabolite
33%
calcium activated,phospholipid dependent protein kinase
33%
Pharmacology, Toxicology and Pharmaceutical Science
Non Insulin Dependent Diabetes Mellitus
100%
Insulin Resistance
66%
Fatty Acid
66%
Tumor Necrosis Factor
33%
calcium activated,phospholipid dependent protein kinase
33%
Obesity
33%
Hyperglycemia
33%
Ceramide
33%
Adiponectin
33%
Diacylglycerol
33%
Adipocytokine
33%