Nitric oxide-dependent bone marrow progenitor mobilization by carbon monoxide enhances endothelial repair after vascular injury

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Abstract

Carbon monoxide (CO) has emerged as a vascular homeostatic molecule that prevents balloon angioplasty-induced stenosis via antiproliferative effects on vascular smooth muscle cells. The effects of CO on reendothelialization have not been evaluated.

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Original languageEnglish
Pages (from-to)537-548
Number of pages12
JournalCirculation
Volume121
Issue number4
DOIs
Publication statusPublished - Feb 2010

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© 2010 American Heart Association, Inc.

    Keywords

  • angioplasty, endothelium, nitric oxide synthase, signal transduction, stenosis

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